Claim · model-written summary
Coinfection suppressed some genes involved in basal resistance responses.
- Taxon
- tobacco
Nicotiana tabacume1 - Treatment
- TH co-infectione0
- Interactor
- TSWV
Orthotospovirus tomatomaculaet0pathogen - Second interactor
- HCRVt0pathogen
- Outcome
- some genes that are involved in these basal resistance responsese0
- Direction
- suppresse0
Not reportedComparator
Verbatim
e0“TH co-infection could not trigger and even suppress some genes that are involved in these basal resistance responses”
t0“tomato spotted wilt orthotospovirus (TSWV) and Hippeastrum chlorotic ringspot orthotospovirus (HCRV)”
e1“TSWV infected tobacco ( Nicotiana benthamiana ) plants”
Title
Min Gui; Huaran Hu; Zhiqiang Jia; et al. Frontiers in Plant Science 2023. doi:10.3389/fpls.2023.1108552
Other claims from this paper (7)
- Association TSWV and HCRV cooperation increased infection efficiency of both viruses in tobacco.
- Intervention effect Individual TSWV infection triggered jasmonic acid signaling, autophagy, and secondary metabolism defense responses.
- Intervention effect Individual HCRV infection triggered jasmonic acid signaling, autophagy, and secondary metabolism defense responses.
- Mechanism The TH complex inhibited NbPR1 expression to suppress tobacco resistance.
- Observation The TH complex altered plant microRNA expression.
- Mechanism Novel-m0782-3p and miR1992-3p interacted with NbSAM and NbWRKY6 and suppressed their expression.
- Mechanism The microRNA mediated suppression led to NbPR1 downregulation and loss of tobacco resistance to both viruses.