Claim · model-written summary
The loss-of-function AGAP mutant had a defective Golgi apparatus with thin and compact cisternae.
- Treatment
- loss-of-function AGAP mutant produced by CRISPR/Cas9e2
- Gene
- AGAPe2
- Outcome
- Golgi apparatuse2
- Direction
- defectivee2
- Qualifier
- with thin and compact cisternaee2
Zea maysNot reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValue
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“ear inflorescences of maize (Zea mays L.)”
e2“The loss-of-function AGAP mutant produced by CRISPR/Cas9 resulted in defective Golgi apparatus with thin and compact cisternae, together with delayed internalization and repressed vesicle agglomeration, leading to defective inflorescences and roots, and dwarfed plants with small leaves”
Title
Manfei Li; Ran Zhao; Yanfang Du; et al. Cells 2021. doi:10.3390/cells10102601
Other claims from this paper (10)
- Intervention effect The loss-of-function AGAP mutant had delayed internalization.
- Intervention effect The loss-of-function AGAP mutant repressed vesicle agglomeration.
- Intervention effect The loss-of-function AGAP mutant had defective inflorescences and roots.
- Intervention effect The loss-of-function AGAP mutant produced dwarfed plants with small leaves.
- Observation AGAP was preferentially localized to the Golgi apparatus.
- Intervention effect The weak agap mutant was phenotypically similar to knr6.
- Intervention effect The weak agap mutant had short ears.
- Intervention effect The weak agap mutant had fewer kernels.
- Observation AGAP interacted with KNR6.
- Intervention effect The agap knr6 double mutant had shorter inflorescence meristems and mature ears than the single mutants.