Claim · model-written summary
Differences in Frankia traits may explain alder host choice and successional shifts.
- Outcome
- host choice and explain successional shiftse0
- Qualifier
- Differences among Frankia OTUs in traits relevant to plant performancee0
Not reportedMaterialGeneCompoundOrgan / stageSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValue
Organisms pictured · Representative pictures of the matched species, not figures from the paper.

Verbatim
e0“Differences among Frankia OTUs in traits relevant to plant performance may form the basis for host choice and explain successional shifts in alder– Frankia assemblages”
Title
Roger W. Ruess; Michael D. Anderson; Jack M. McFarland; et al. Ecological Monographs 2012. doi:10.1890/12-0782.1
Other claims from this paper (14)
- Intervention effect P fertilization increased alder nodule biomass relative to controls.
- Intervention effect P fertilization increased alder N-fixation rates relative to controls.
- Intervention effect N fertilization lowered alder nodule biomass relative to controls.
- Intervention effect N fertilization lowered alder N-fixation rates relative to controls.
- Intervention effect Stand-level N inputs in +P plots exceeded +N plots by more than an order of magnitude.
- Association Nodule respiration and N-fixation rates were positively correlated in alder.
- Mechanism Alder increased carbon use efficiency when N fixation was up-regulated.
- Observation Two of eight identified Frankia OTUs were dominant.
- Association Fertilization explained significant differences in Frankia OTU composition.
- Observation Dominant Frankia OTUs responded similarly to up- and down-regulation treatments.
- Observation Dominant Frankia OTUs differed in carbon costs of N fixation.
- Intervention effect The most abundant subdominant Frankia OTU failed to increase N fixation in +P plots.
- Mechanism Manipulating Frankia assemblages was suggested as an alder adaptation across different environments.
- Mechanism Alder changes in carbon allocation to nodules appear to drive and respond to environmental heterogeneity.