Claim · model-written summary
Treated F. sacchari conidial membranes had irregular structure and uneven thickness.
- Taxon
- sugarcanet0
- Interactor
- F. sacchariFusarium saccharie1pathogen
- Outcome
- cell membrane showed irregular structure and no clear discernable level, with uneven thicknesse0
Not reportedMaterialGeneCompoundOrgan / stageSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValueQualifier
Verbatim
e0“the cell membrane showed irregular structure and no clear discernable level, with uneven thickness”
e1“conidia of F. sacchari”
t0“sugarcane”
Title
Aiming Jiang; Xiang Xu; Zunwei Ke; et al. Pest Management Science 2026. doi:10.1002/ps.70415
Other claims from this paper (10)
- Intervention effect Fusaricidin A suppressed germination of F. sacchari conidia.
- Intervention effect Fusaricidin A protected sugarcane against pokkah boeng disease in glasshouse assays.
- Observation Treated F. sacchari conidia were poorly stacked, ruptured or collapsed.
- Intervention effect Treated cells accumulated significantly more liposomes.
- Intervention effect Fusaricidin A disrupted F. sacchari plasma membrane permeability.
- Intervention effect Fusaricidin A enhanced spore apoptosis.
- Intervention effect Fusaricidin A caused excessive mitochondrial reactive oxygen species production.
- Observation Antioxidant genes GSH1, GSH2, SOD1 and SOD2 were upregulated in treated conidia.
- Observation Mkk1, Mpk1 and Swi4 were downregulated in treated conidia.
- Mechanism The authors infer that fusaricidin A impairs conidial wall stability through membrane damage, ROS stress and wall integrity regulation.