Claim · model-written summary
The authors suggested both aphid species spread mosaic equally.
- Taxon
- sugar‐beet
Beta vulgaris subsp. vulgarist0 - Interactor
- M. persicae
Myzus persicaee0pestt1 - Second interactor
- A. fabae
Aphis fabaee0pestt1 - Outcome
- spread the viruse0
Not reportedMaterialGeneCompoundOrgan / stageTreatmentComparatorSettingDirectionValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.



Verbatim
e0“there is a strong suggestion that alatae of M. persicae and A. fabae spread the virus, and that the contributions of both species are equal.”
t0“THE SPREAD OF BEET YELLOWS AND BEET MOSAIC VIRUSES IN THE SUGAR‐BEET ROOT CROP”
t1“THE SPREAD OF BEET YELLOWS AND BEET MOSAIC VIRUSES IN THE SUGAR‐BEET ROOT CROP”
Title
THE SPREAD OF BEET YELLOWS AND BEET MOSAIC VIRUSES IN THE SUGAR‐BEET ROOT CROP
MARION A. WATSON; M. J. R. HEALY Annals of Applied Biology 1953. doi:10.1111/j.1744-7348.1953.tb02365.x
Other claims from this paper (13)
- Association Spring and summer alate Myzus persicae were the strongest predictor of beet yellows spread.
- Observation Apterous M. persicae spread little beet yellows virus.
- Observation Alate and apterous Aphis fabae contributed negligibly to beet yellows spread.
- Method / resource A spread model adequately fit observed beet yellows infection using trap counts and assumed aphid parameters.
- Observation Yellows spread mainly within or among nearby fields after spring introduction.
- Observation Summer migrants from infected seed crops spread some infection to root crops in intensive seed crop areas.
- Mechanism Greater yellows prevalence in seed crop areas was attributed to greater M. persicae infestation.
- Observation The proportion infected by spring migrants before June end was small and variable.
- Association Initial infection did not differ significantly between seed and nonseed areas.
- Observation Mosaic spread mainly near seed crops within seed crop areas.
- Association No significant aphid number relation with mosaic infection increase was established.
- Mechanism The authors inferred mosaic spread from outside migrants instead of movement inside root crops.
- Mechanism Variation in seed crop infection and migrant origin was proposed to explain irregular mosaic incidence data.