Claim · model-written summary
OsGRX6 interacted with OsbZIP83.
- Taxon
- rice
Oryza sativat0 - Gene
- OsGRX6e0
- Outcome
- interacted with OsbZIP83e0
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
t0“rice”
e0“OsGRX6 also interacted with OsbZIP83”
Title
KOBAYASHI,Takanori; SHINKAWA,Haruka; NAGANO,Atsushi J; et al. Institutional Repositories DataBase (IRDB)
Other claims from this paper (14)
- Observation Iron deficiency or OsHRZ knockdown produced differential regulation between transcript and protein levels for many genes, including OsbZIP83.
- Observation OsGRX6 interacted with OsHRZ proteins in yeast and plant cells.
- Observation OsGRX9 interacted with OsHRZ proteins in yeast and plant cells.
- Observation In vitro assays suggested OsbZIP83 undergoes 26S proteasome- and OsHRZ-dependent degradation.
- Observation In vitro assays suggested OsGRX6 undergoes 26S proteasome- and OsHRZ-dependent degradation.
- Observation In vitro assays suggested OsGRX9 undergoes 26S proteasome- and OsHRZ-dependent degradation.
- Observation OsbZIP83 protein was preferentially degraded in iron-deficient rice roots.
- Intervention effect OsGRX9 overexpression improved rice tolerance to iron deficiency.
- Intervention effect OsbZIP83 overexpression improved rice tolerance to iron deficiency.
- Observation OsGRX9 knockdown affected expression of iron-related genes.
- Observation OsGRX6 knockdown affected expression of iron-related genes.
- Intervention effect OsbZIP83 overexpression enhanced expression of OsYSL2 and OsNAS3.
- Intervention effect OsbZIP83 overexpression enhanced expression of OsGRX9 and genes related to phytoalexin biosynthesis and the salicylic acid pathway.
- Mechanism The authors infer that OsbZIP83, OsGRX6 and OsGRX9 facilitate iron utilization downstream of OsHRZ.