Claim · model-written summary
ZmbZIP92 is highly homologous to the rice gene OsbZIP62.
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentSettingDirectionValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.

Verbatim
e0“This gene is highly homologous to the rice (Oryza sativa L.) gene OsbZIP62”
t0“Maize ZmbZIP92”
Title
Maize ZmbZIP92 transcription factor positively regulates drought tolerance in Arabidopsis
Zukuan Liu; Jian Shang; Min Liu; et al. Plant Signaling & Behavior 2026. doi:10.1080/15592324.2026.2635681
Other claims from this paper (13)
- Observation The drought response-related candidate gene ZmbZIP92 was cloned from maize following a comparative genomic analysis.
- Observation ZmbZIP92 exhibits tissue-specific expression patterns.
- Intervention effect ZmbZIP92 expression was significantly induced by drought.
- Intervention effect ZmbZIP92 expression was significantly induced by high salinity.
- Intervention effect ZmbZIP92 expression was significantly induced by abscisic acid treatment.
- Observation ZmbZIP92 is a nuclear protein.
- Observation Yeast-based assays showed ZmbZIP92 lacks transcriptional self-activation.
- Observation Dual-luciferase reporter assays showed ZmbZIP92 binds specifically to the G-box (CACGTG) cis-element.
- Intervention effect Overexpressing ZmbZIP92 in Arabidopsis thaliana significantly promoted root elongation.
- Intervention effect Overexpressing ZmbZIP92 in Arabidopsis thaliana enhanced drought tolerance.
- Intervention effect Overexpressing ZmbZIP92 in Arabidopsis thaliana increased sensitivity to ABA, reflected by markedly inhibited seed germination.
- Observation RNA sequencing indicated that multiple stress response-related pathways, including ABA signalling, antioxidant response, transmembrane transport and general signal transduction, were enriched in ZmbZIP92-overexpressing plants.
- Mechanism ZmbZIP92 positively regulates drought tolerance through effects on the ABA signalling pathway and other stress response-related signalling networks.