Claim · model-written summary
AHK significantly increased NtCOI1 transcription in inoculated and systemic leaves.
- Taxon
- tobacco plants
Nicotiana tabacumt0 - Organ / stage
- inoculated and systemic leavese0
- Treatment
- AHK-treatede0
- Gene
- NtCOI1e0
- Outcome
- transcription of the NtCOI1 genee0
- Direction
- highere0
- Comparator
- controle0
Not reportedMaterialCompoundInteractorInteractor roleSecond interactorSecond interactor roleSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“The transcription of the NtCOI1 gene, a key gene in the JA pathway, was significantly higher in both the inoculated and systemic leaves of the AHK-treated plants compared to the control”
t0“tobacco plants”
Title
Lihua Zhao; Zhonghui Hu; Shunlin Li; et al. Scientific Reports 2019. doi:10.1038/s41598-019-39247-6
Other claims from this paper (9)
- Intervention effect AHK had greater curative effects against TSWV than ningnanmycin.
- Intervention effect AHK had greater protective effects against TSWV than ningnanmycin.
- Intervention effect AHK delayed initiation of expression of all TSWV genes by over one day in the curative assay.
- Intervention effect AHK inhibited expression of NSs, NSm and RdRp viral genes.
- Intervention effect AHK inhibited replication of all TSWV genes in systemic leaves by over 90%.
- Intervention effect AHK increased jasmonic acid concentrations in treated and systemic leaves.
- Intervention effect AHK increased jasmonic acid isoleucine concentrations in treated and systemic leaves.
- Mechanism AHK was inferred to induce systemic resistance.
- Intervention effect Silencing NtCOI1 eliminated AHK-induced TSWV resistance in Nicotiana benthamiana.