Claim · model-written summary
LOB1, LOB2, and DIOX expression differed between Pera and Tahiti plants with PthA dependence.
- Material
- 'Pera'e0
- Gene
- LOB1, LOB2 and DIOXe0
- Outcome
- PthA-dependent expressione0
- Direction
- differentiale0
- Comparator
- 'Tahiti'e0
CitrusNot reportedCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“LOB1, LOB2 and DIOX show differential PthA-dependent expression between 'Pera' and 'Tahiti' plants”
t0“citrus”
Title
Valeria Yukari Abe; Celso Eduardo Benedetti Molecular Plant Pathology 2015. doi:10.1111/mpp.12359
Other claims from this paper (11)
- Observation PthA4 was indispensable for canker formation across six Brazilian citrus varieties.
- Observation PthAs 1 and 3 contributed to canker development in Pera sweet orange.
- Observation PthAs 1 and 3 did not contribute to canker development in Tahiti lemon.
- Intervention effect Deleting two or more pthA genes reduced bacterial growth in planta more than single deletions.
- Association PthA1 and PthA3 contributions to Pera cankers did not correlate with LOB1 activation.
- Association PthA1 and PthA3 contributions to Pera cankers correlated with induction of LOB2 and DIOX.
- Association PthA-dependent expression differences appeared associated with nucleotide polymorphisms near PthA-binding sites.
- Mechanism LOB1 activation alone was insufficient to cause citrus cankers.
- Mechanism DIOX acted as a canker susceptibility gene in Pera plants.
- Mechanism DIOX did not act as a canker susceptibility gene in Tahiti plants.
- Mechanism The authors infer that activation of multiple susceptibility genes is needed for full canker development.