Claim · model-written summary
At pH 2.2 the pathogen shows shortened hyphae.
- Treatment
- pH 2.2e3
- Interactor
- Geotrichum citri-aurantiiGeotrichum citri-aurantiie1pathogen
- Outcome
- hyphaee3
- Direction
- shortenede3
CitrusNot reportedMaterialGeneCompoundOrgan / stageSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e1“How the pathogen, Geotrichum citri-aurantii, adapts to the highly acidic environment of citrus fruit remains inadequately understood”
e3“It exhibited strong adaptability at pH 2.2, showing shortened and aggregated hyphae, delayed spore germination, and increased vacuoles”
Title
Insights into the Adaptation of Geotrichum citri-aurrantii in Highly Acidic Environments
Qian Niu; Jie Zheng; Wenbin Liao; et al. Journal of Fungi 2026. doi:10.3390/jof12020091
Other claims from this paper (10)
- Observation G. citri-aurantii alkalizes (pH below 3.00) or acidifies (pH above 3.00) the citrus host tissue.
- Observation At pH 2.2 the pathogen shows delayed spore germination.
- Observation At pH 2.2 the pathogen shows increased vacuoles.
- Observation Transcriptomics and qRT-PCR show significant regulation of pathogen genes involved in cell wall remodeling under acidic conditions.
- Observation Transcriptomics and qRT-PCR show significant regulation of pathogen genes involved in cell membrane component synthesis under acidic conditions.
- Observation Transcriptomics and qRT-PCR show significant regulation of pathogen genes involved in carbon metabolism under acidic conditions.
- Observation Transcriptomics and qRT-PCR show significant regulation of pathogen genes involved in signal transduction under acidic conditions.
- Mechanism The regulatory changes are proposed to let the pathogen block an influx of external acids.
- Mechanism The regulatory changes are proposed to let the pathogen maintain its energy supply under acid stress.
- Observation Pal/Rim pH signaling pathway genes of the pathogen respond with distinct patterns in citrus cultivars with different acidities.