Claim · model-written summary
Antisera from the pathogenic types revealed cross-reactions with P. graminis avenae.
- Interactor
- P. graminis avenae
Puccinia graminise2pathogen - Outcome
- cross‐reactionse2
Zea maysNot reportedMaterialGeneCompoundOrgan / stageSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“scored for pathogenicity on 6 inbred lines of maize”
e1“Four urediospore clones of P. sorghi were inbred”
e2“Antisera produced from the pathogenic types differentiated the homologous from the heterologous combinations and revealed cross‐reactions with P. graminis avenae”
Title
THE GENETIC CONTROL OF PATHOGENICITY, SEROTYPES AND VARIABILITY IN PUCCINIA SORGHI
Arthur L. Flangas; James G. Dickson American Journal of Botany 1961. doi:10.1002/j.1537-2197.1961.tb11638.x
Other claims from this paper (8)
- Observation Inbred recombinant lines of P. sorghi showed similar changes from original pathogenicity, an ultimate gain or loss in host range or specificity.
- Observation Continued inbreeding did not segregate homozygous pathogenic complements into distinct pathogenic biotypes.
- Observation A similar array of pathogenic types was produced after each generation of inbreeding.
- Observation Suppressed pathogenicity types in the dicaryotic line were revealed only through recombination.
- Observation Hybridization confirmed the indeterminate nature of the pathogenic locus in P. sorghi.
- Mechanism The mesothetic-X reaction is explained by heterologous complementarity of fit of the interacting genic systems.
- Observation Heterologous mesothetic-X reactions cannot be explained on the discrete gene-for-gene basis of rust reaction.
- Association A possible parallel exists between the modes of inheritance of pathogenic specificity and antigenic specificity in P. sorghi.