Claim · model-written summary
Heterologous mesothetic-X reactions cannot be explained on the discrete gene-for-gene basis of rust reaction.
- Outcome
- heterologous mesothetic‐X reactionse2
- Qualifier
- cannot be explained on the discrete gene‐for‐gene basis of rust reactione2
Zea maysNot reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValue
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“scored for pathogenicity on 6 inbred lines of maize”
e1“Four urediospore clones of P. sorghi were inbred”
e2“The heterologous mesothetic‐X reactions offer ambiguous definition to the specificity state of the interaction and cannot be explained on the discrete gene‐for‐gene basis of rust reaction”
Title
THE GENETIC CONTROL OF PATHOGENICITY, SEROTYPES AND VARIABILITY IN PUCCINIA SORGHI
Arthur L. Flangas; James G. Dickson American Journal of Botany 1961. doi:10.1002/j.1537-2197.1961.tb11638.x
Other claims from this paper (8)
- Observation Inbred recombinant lines of P. sorghi showed similar changes from original pathogenicity, an ultimate gain or loss in host range or specificity.
- Observation Continued inbreeding did not segregate homozygous pathogenic complements into distinct pathogenic biotypes.
- Observation A similar array of pathogenic types was produced after each generation of inbreeding.
- Observation Suppressed pathogenicity types in the dicaryotic line were revealed only through recombination.
- Observation Hybridization confirmed the indeterminate nature of the pathogenic locus in P. sorghi.
- Mechanism The mesothetic-X reaction is explained by heterologous complementarity of fit of the interacting genic systems.
- Observation Antisera from the pathogenic types revealed cross-reactions with P. graminis avenae.
- Association A possible parallel exists between the modes of inheritance of pathogenic specificity and antigenic specificity in P. sorghi.