Claim · model-written summary
PvRxLR16 significantly enhanced resistance to Phytophthora capsici.
- Taxon
- Nicotiana benthamiana
Nicotiana benthamianat0 - Treatment
- PvRxLR16e0
- Interactor
- Phytophthora capsici
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Phytophthora capsicie0pathogen - Outcome
- plant resistance to Phytophthora capsicie0
- Direction
- enhancee0
Not reportedMaterialGeneCompoundOrgan / stageSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
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Verbatim
e0“PvRxLR16 could also significantly enhance plant resistance to Phytophthora capsici and the nuclear localization was required for this ability.”
t0“Nicotiana benthamiana”
Title
Jiang Xiang; Xinlong Li; Ling Yin; et al. BMC Plant Biology 2017. doi:10.1186/s12870-017-1016-4
Other claims from this paper (6)
- Mechanism PvRxLR16 nuclear localization, W/Y/L motifs and glycosylation site were essential to its cell death activity.
- Mechanism SGT1, HSP90 and RAR1 were required for the PvRxLR16 cell death response.
- Mechanism SERK3 was not required for the PvRxLR16 cell death response.
- Mechanism Mitogen activated kinases and transcription factors were involved in PvRxLR16 perception.
- Mechanism PvRxLR16 nuclear localization was required for enhanced resistance to P. capsici.
- Intervention effect Other PvRxLR effectors suppressed PvRxLR16 induced defense and disease resistance.