Claim · model-written summary
Resting membrane potential was about −53 mV, agreeing with microelectrode measurements of about −55 mV.
Not reportedTaxonMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingDirection
Verbatim
e0“The resting membrane potential (Em) according to our measurements with TPP+ is about −53 mV and is in agreement with membrane potential values as measured with intracellular microelectrodes (about −55 mV).”
Title
Sjoukje Heimovaara-Dijkstra; Bert van Duijn; Kees R. Libbenga; et al. Plant and Cell Physiology 1994. doi:10.1093/oxfordjournals.pcp.a078652
Other claims from this paper (10)
- Association Resting membrane potential depended on external pH.
- Intervention effect ABA transiently hyperpolarized the membrane to −62 mV.
- Association Hyperpolarization depended on ABA concentration.
- Intervention effect DES prevented hyperpolarization.
- Intervention effect Micanozole prevented hyperpolarization.
- Mechanism Hyperpolarization was interpreted as consistent with H+-ATPase activation.
- Intervention effect BaCl2 prolonged hyperpolarization.
- Mechanism Hyperpolarization was suggested to open K+ channels.
- Intervention effect DES inhibited ABA-induced gene expression.
- Intervention effect BaCl2 had a slight inhibitory effect on ABA-induced gene expression.