Claim · model-written summary
Illumination caused photoinhibition at every studied light intensity.
- Taxon
- Pumpkine0
- Treatment
- illumination at all light intensitiese0
- Outcome
- ratio of variable to maximum fluorescencee0
- Direction
- decreasee0
- Qualifier
- presence of lincomycine0
Not reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValue
Verbatim
e0“Pumpkin leaves grown under high light (500-700 micromol of photons m-2.s-1) were illuminated under photon flux densities ranging from 6.5 to 1500 micromol.m-2.s-1 in the presence of lincomycin, an inhibitor of chloroplast protein synthesis. The illumination at all light intensities caused photoinhibition, measured as a decrease in the ratio of variable to maximum fluorescence.”
Title
E Tyystjärvi; E M Aro Proceedings of the National Academy of Sciences 1996. doi:10.1073/pnas.93.5.2213
Other claims from this paper (10)
- Association Loss of PSII electron transfer activity correlated with declining fluorescence ratio.
- Association The photoinhibition rate constant was directly proportional to photon flux density.
- Observation The fluorescence ratio did not decrease under low light without lincomycin.
- Observation The fluorescence ratio did not decrease in darkness with lincomycin.
- Observation Quantum yield of photoinhibition remained constant across photon flux densities.
- Mechanism The authors infer one dominant in vivo photoinhibition mechanism across light intensities.
- Mechanism The authors consider the in vivo mechanism probably distinct from the anaerobic acceptor-side mechanism.
- Association Loss of PSII activity correlated strongly with D1 protein loss under low light.
- Observation Inactive PSII centers with D1 awaiting degradation did not accumulate under low light.
- Mechanism D1 protein turnover was inferred to depend on photoinhibition at low and high light.