Claim · model-written summary
Heat stress increased senescence.
- Taxon
- Tomato
Solanum lycopersicumt0 - Treatment
- Heat stresse0
- Outcome
- senescencee0
- Direction
- acceleratede0
Not reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
t0“Tomato”
e0“Heat stress significantly accelerated senescence, damage to the photosystem and upregulation of reactive oxygen species (ROS), generating RBOH gene expression.”
Title
Mohammad Shah Jahan; Sheng Shu; Yu Wang; et al. Frontiers in Plant Science 2021. doi:10.3389/fpls.2021.650955
Other claims from this paper (15)
- Intervention effect Heat stress increased damage to the photosystem.
- Intervention effect Heat stress increased reactive oxygen species (ROS).
- Intervention effect Melatonin improved leaf senescence under heat stress.
- Intervention effect Melatonin improved leaf yellowing under heat stress.
- Intervention effect Melatonin improved Fv/Fm ratio under heat stress.
- Intervention effect Melatonin improved ROS production under heat stress.
- Intervention effect MT suppressed Rbohs, chlorophyll catabolic, and senescence-associated gene expression.
- Intervention effect MT application changed endogenous MT in heat-exposed tomato.
- Intervention effect MT application changed GA contents in heat-exposed tomato.
- Intervention effect MT application changed ABA content in heat-exposed tomato.
- Intervention effect Inhibitors compromised MT-induced heat tolerance.
- Intervention effect MT altered ABA biosynthesis and signaling genes.
- Intervention effect MT altered biosynthesis genes MT and GA.
- Intervention effect MT altered GA signaling suppressor and catabolic gene expression.
- Intervention effect MT altered ABA catabolic gene expression.