Claim · model-written summary
AtGCN4 overexpression reduced pathogen entry.
- Taxon
- Arabidopsis thaliana
Arabidopsis thalianat0 - Treatment
- AtGCN4 overexpressione0
- Outcome
- pathogen entrye0
- Direction
- reducede0
Not reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“AtGCN4 overexpression plants have reduced H+-ATPase activity, stomata that are less responsive to pathogen virulence factors such as coronatine (phytotoxin produced by the bacterium Pseudomonas syringae) or fusicoccin (a fungal toxin produced by the fungus Fusicoccum amygdali), reduced pathogen entry”
t0“Arabidopsis thaliana”
Title
Amita Kaundal; Vemanna S. Ramu; Sunhee Oh; et al. The Plant Cell 2017. doi:10.1105/tpc.17.00070
Other claims from this paper (8)
- Intervention effect GCN4 silencing compromised host and nonhost resistance during pathogen infection in both plants.
- Intervention effect GCN4 silencing left stomata open during pathogen infection.
- Intervention effect AtGCN4 overexpression reduced H+-ATPase activity.
- Intervention effect AtGCN4 overexpression reduced stomatal responsiveness to coronatine.
- Intervention effect AtGCN4 overexpression reduced stomatal responsiveness to fusicoccin.
- Intervention effect AtGCN4 overexpression enhanced drought tolerance.
- Observation AtGCN4 interacted with RIN4 and 14-3-3 proteins.
- Mechanism Authors propose GCN4 degrades RIN4 and 14-3-3 through a proteasome-mediated pathway to reduce proton pump activity and close stomata.