Claim · model-written summary
Interference with ATG8 inhibited pericarp programmed cell death.
- Taxon
- Wheat
Triticum aestivumt0 - Treatment
- interference of the key autophagy gene ATG8e0
- Gene
- ATG8e0
- Outcome
- PCDe0
- Direction
- inhibitede0
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“following interference of the key autophagy gene ATG8 , PCD was inhibited and the thickness of the pericarp increased, resulting in small premature grains”
t0“Wheat Grains”
Title
Programmed Degradation of Pericarp Cells in Wheat Grains Depends on Autophagy
Yong-Bo Li; Mei Yan; De-Zhou Cui; et al. Frontiers in Genetics 2021. doi:10.3389/fgene.2021.784545
Other claims from this paper (9)
- Observation Pericarp was thinnest at the spike top, intermediate in the middle and thickest at the bottom.
- Mechanism Pericarp cell-layer number corresponded to thickness differences and decreased through programmed cell death.
- Observation ATG8-PE expression increased before autophagy structures appeared.
- Observation Autophagy structures appeared following increased ATG8-PE expression.
- Intervention effect Interference with ATG8 increased pericarp thickness.
- Intervention effect ATG8 interference resulted in small premature grains.
- Observation Autophagy and programmed cell death coexist during early wheat pericarp development.
- Observation Autophagy and PCD increased from spike bottom to top.
- Mechanism Pericarp PCD depends on ATG8-mediated autophagy.