Claim · model-written summary
OsDIR55 disruption caused excessive phosphate accumulation under sufficient phosphate.
- Taxon
- Rice
Oryza sativat0 - Gene
- OsDIR55e0
- Outcome
- P i accumulatione0
- Direction
- excessivee0
- Qualifier
- P i ‐sufficient conditionse0
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingValue
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“Disruption of OsDIR55 leads to excessive P i accumulation and aberrant physiological responses under P i ‐sufficient conditions with leaf tip necrosis, indicative of P i toxicity.”
t0“Rice”
Title
OsPHR2‐OsDIR55 Module Enhances Root Barrier Integrity and Rice Tolerance to Phosphate Deficiency
Wen Duan; Xuemei Li; Baoping Xue; et al. Plant Cell & Environment 2026. doi:10.1111/pce.70686
Other claims from this paper (7)
- Observation Phosphate deficiency induced localized lignification in rice roots.
- Intervention effect OsDIR55 overexpression improved plant performance under low phosphate, including root-to-shoot ratio and biomass.
- Observation Loss-of-function dir55 mutants showed contrasting phenotypes under low phosphate.
- Observation OsPHR2 binds P1BS elements in the OsDIR55 promoter.
- Observation Phosphate deprivation upregulated OsDIR55 expression dependent on OsPHR2.
- Mechanism OsDIR55 maintained cellular phosphate homeostasis under starvation by reinforcing the root apoplastic barrier.
- Observation OsDIR55 disruption caused leaf tip necrosis under sufficient phosphate.