Claim · model-written summary
The B component may have evolved from the 180-bp knob repeat through mutations during evolution (author interpretation).
Not reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValue
Organisms pictured · Representative pictures of the matched species, not figures from the paper.

Verbatim
e2“we isolated 59 clones belonging to the TR-1 family from maize and teosinte”
e3“the B component may evolve from the 180-bp knob repeat as a result of mutations during evolution”
Title
F C Hsu; C J Wang; C M Chen; et al. Genetics 2003. doi:10.1093/genetics/164.3.1087
Other claims from this paper (8)
- Observation TR-1 family members are composed of three basic sequences (A, B or B', 2/3B or 2/3B', C) arranged in combinations that give repeat units of about 180 bp multiples.
- Mechanism B' may have originated from B through lateral amplification accompanied by base-pair changes (author interpretation).
- Mechanism C plus A may be a single sequence added to B and B', probably via nonhomologous recombination (author interpretation).
- Mechanism 69 bp at the 3' end of B or B' and the entire sequence of C can be removed from the elements by an unknown mechanism (author interpretation).
- Observation TR-1 elements show partial sequence homology with two centromeric B-repeat sequences of the supernumerary B chromosome.
- Mechanism The B repeat may be derived from knob repeats in A chromosomes, which then became structurally modified (author interpretation).
- Observation Fluorescence in situ hybridization localized the B repeat to the B centromere.
- Observation Fluorescence in situ hybridization localized the 180-bp and TR-1 repeats to the proximal heterochromatin knob on the B chromosome.