Claim · model-written summary
Sugar effect was independent of TOR.
- Taxon
- Arabidopsis
Arabidopsise1 - Gene
- Target of Rapamycin (TOR)e0
- Outcome
- independent of the sugar sensorse0
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“This sugar effect is independent of the sugar sensors Hexokinase 1 (HXK1) and Target of Rapamycin (TOR)”
e1“sugar inhibits BR signaling in Arabidopsis seedlings grown under light”
Title
Sugar inhibits brassinosteroid signaling by enhancing BIN2 phosphorylation of BZR1
Zhenzhen Zhang; Ying Sun; Xue Jiang; et al. PLoS Genetics 2021. doi:10.1371/journal.pgen.1009540
Other claims from this paper (7)
- Intervention effect Sugar inhibited BR signaling in light-grown Arabidopsis seedlings.
- Intervention effect Increasing sucrose inhibited BR-induced hypocotyl elongation in light-grown seedlings.
- Association Sugar inhibition correlated with a reduced BR effect on BZR1 dephosphorylation.
- Observation Sugar effect was independent of HXK1.
- Mechanism The sugar effect required BIN2.
- Intervention effect Sugar stabilized BIN2.
- Mechanism Authors suggest light-dependent sugar-BR crosstalk helps optimize growth responses to photosynthate availability.