Claim · model-written summary
Autotetraploidization increased Alternaria resistance.
- Taxon
- Chrysanthemum lavandulifolium
Chrysanthemum lavandulifoliume0 - Treatment
- autotetraploidizatione0
- Interactor
- Alternaria alternata
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Alternaria alternatae0pathogen - Outcome
- resistance to Alternaria alternatae0
- Direction
- enhancese0
Not reportedMaterialGeneCompoundOrgan / stageSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
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Verbatim
e0“autotetraploidization in Chrysanthemum lavandulifolium significantly enhances resistance to Alternaria alternata”
Title
Zhongyu Yu; Huiting Ci; Ruyue Jing; et al. Horticulture Research 2026. doi:10.1093/hr/uhag050
Other claims from this paper (6)
- Observation Polyploidization caused promoter CHH hypomethylation in WRKY factors.
- Mechanism WRKY transcription was activated upon fungal infection.
- Mechanism CIWRKY103 was important for disease resistance.
- Intervention effect 5-azacytidine activated WRKY103 expression in diploids.
- Intervention effect 5-azacytidine increased resistance in diploids.
- Observation The epigenetic mechanism was conserved across chrysanthemum species.