Chrysanthemum lavandulifolium 177 claims

species · family Asteraceae · green plant · NCBI Taxonomy ↗

Also written in claims: Dendranthema lavandulifolium · D. lavandulifolium · D.lavandulifolium · C. lavandulifolium Makino — NCBI synonyms: Dendranthema lavandulifolium · Pyrethrum lavandulifolium

7 spellings

Chrysanthemum lavandulifolium · picture from its Wikimedia Commons categoryGrrewa · CC BY-SA 4.0 · Wikimedia Commons ↗

177 claims

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Intervention effectobservedoriginal resultpositivesignificant (as stated)measured

Claim · model-written summary

Autotetraploidization increased Alternaria resistance.

Taxon: Chrysanthemum lavandulifoliumChrysanthemum lavandulifoliume0
Treatment: autotetraploidizatione0
Interactor: Alternaria alternataAlternaria alternatae0pathogen
Outcome: resistance to Alternaria alternatae0
Direction: enhancese0

Not reportedComparator

Verbatim

e0
“autotetraploidization in Chrysanthemum lavandulifolium significantly enhances resistance to Alternaria alternata”
✓ located verbatim in the source · abstract, characters 224–336

Title

Polyploidization enhances plant resistance to Alternaria alternata via DNA hypomethylation activated WRKYs

Zhongyu Yu; Huiting Ci; Ruyue Jing; et al. Horticulture Research 2026. doi:10.1093/hr/uhag050

Other claims from this paper (6)

Observation Polyploidization caused promoter CHH hypomethylation in WRKY factors.
Mechanism WRKY transcription was activated upon fungal infection.
Mechanism CIWRKY103 was important for disease resistance.
Intervention effect 5-azacytidine activated WRKY103 expression in diploids.
Intervention effect 5-azacytidine increased resistance in diploids.
Observation The epigenetic mechanism was conserved across chrysanthemum species.

Observationobservedoriginal resultmeasured

Claim · model-written summary

Polyploidization caused promoter CHH hypomethylation in WRKY factors.

Taxon: Chrysanthemum lavandulifoliumChrysanthemum lavandulifoliumt0
Gene: WRKY transcription factorse0
Outcome: CHH hypomethylatione0

Not reportedDirection

Verbatim

e0
“polyploidization induces locus-specific CHH hypomethylation in the promoters of a subset of WRKY transcription factors”
✓ located verbatim in the source · abstract, characters 433–551

t0
“in Chrysanthemum lavandulifolium”
✓ located verbatim in the source · abstract, characters 246–278

Title

Polyploidization enhances plant resistance to Alternaria alternata via DNA hypomethylation activated WRKYs

Zhongyu Yu; Huiting Ci; Ruyue Jing; et al. Horticulture Research 2026. doi:10.1093/hr/uhag050

Other claims from this paper (6)

Intervention effect Autotetraploidization increased Alternaria resistance.
Mechanism WRKY transcription was activated upon fungal infection.
Mechanism CIWRKY103 was important for disease resistance.
Intervention effect 5-azacytidine activated WRKY103 expression in diploids.
Intervention effect 5-azacytidine increased resistance in diploids.
Observation The epigenetic mechanism was conserved across chrysanthemum species.

Mechanismobservedoriginal resultmeasured

Claim · model-written summary

WRKY transcription was activated upon fungal infection.

Taxon: Chrysanthemum lavandulifoliumChrysanthemum lavandulifoliume1
Gene: WRKYst0
Outcome: transcriptional activatione0
Qualifier: upon fungal infectione0

Verbatim

e0
“leading to their transcriptional activation upon fungal infection”
✓ located verbatim in the source · abstract, characters 553–618

t0
“WRKYs”
✓ located verbatim in the source · title, characters 101–106

e1
“autotetraploidization in Chrysanthemum lavandulifolium significantly enhances resistance to Alternaria alternata”
✓ located verbatim in the source · abstract, characters 224–336

Title

Polyploidization enhances plant resistance to Alternaria alternata via DNA hypomethylation activated WRKYs

Zhongyu Yu; Huiting Ci; Ruyue Jing; et al. Horticulture Research 2026. doi:10.1093/hr/uhag050

Other claims from this paper (6)

Intervention effect Autotetraploidization increased Alternaria resistance.
Observation Polyploidization caused promoter CHH hypomethylation in WRKY factors.
Mechanism CIWRKY103 was important for disease resistance.
Intervention effect 5-azacytidine activated WRKY103 expression in diploids.
Intervention effect 5-azacytidine increased resistance in diploids.
Observation The epigenetic mechanism was conserved across chrysanthemum species.

Mechanismobservedoriginal resultmeasured

Claim · model-written summary

CIWRKY103 was important for disease resistance.

Taxon: Chrysanthemum lavandulifoliumChrysanthemum lavandulifoliumt0
Gene: CIWRKY103e0
Outcome: disease resistancee0
Qualifier: critical rolee0

Verbatim

e0
“Functional characterization of CIWRKY103, a key hypomethylated WRKY gene, confirms its critical role in conferring disease resistance.”
✓ located verbatim in the source · abstract, characters 620–754

t0
“in Chrysanthemum lavandulifolium”
✓ located verbatim in the source · abstract, characters 246–278

Title

Polyploidization enhances plant resistance to Alternaria alternata via DNA hypomethylation activated WRKYs

Zhongyu Yu; Huiting Ci; Ruyue Jing; et al. Horticulture Research 2026. doi:10.1093/hr/uhag050

Other claims from this paper (6)

Intervention effect Autotetraploidization increased Alternaria resistance.
Observation Polyploidization caused promoter CHH hypomethylation in WRKY factors.
Mechanism WRKY transcription was activated upon fungal infection.
Intervention effect 5-azacytidine activated WRKY103 expression in diploids.
Intervention effect 5-azacytidine increased resistance in diploids.
Observation The epigenetic mechanism was conserved across chrysanthemum species.

Intervention effectobservedoriginal resultpositivemeasured

Claim · model-written summary

5-azacytidine activated WRKY103 expression in diploids.

Taxon: Chrysanthemum lavandulifoliumChrysanthemum lavandulifoliumt0
Treatment: 5-azacytidine treatmente0
Gene: WRKY103e0
Outcome: WRKY103 expressione0
Direction: activatinge0
Comparator: tetraploid phenotypee0

Verbatim

e0
“Chemical inhibition of DNA methylation (5-azacytidine treatment) in diploid plants mimics the tetraploid phenotype by activating WRKY103 expression and enhancing resistance.”
✓ located verbatim in the source · abstract, characters 755–928

t0
“in Chrysanthemum lavandulifolium”
✓ located verbatim in the source · abstract, characters 246–278

Title

Polyploidization enhances plant resistance to Alternaria alternata via DNA hypomethylation activated WRKYs

Zhongyu Yu; Huiting Ci; Ruyue Jing; et al. Horticulture Research 2026. doi:10.1093/hr/uhag050

Other claims from this paper (6)

Intervention effect Autotetraploidization increased Alternaria resistance.
Observation Polyploidization caused promoter CHH hypomethylation in WRKY factors.
Mechanism WRKY transcription was activated upon fungal infection.
Mechanism CIWRKY103 was important for disease resistance.
Intervention effect 5-azacytidine increased resistance in diploids.
Observation The epigenetic mechanism was conserved across chrysanthemum species.

Intervention effectobservedoriginal resultpositivemeasured

Claim · model-written summary

5-azacytidine increased resistance in diploids.

Taxon: Chrysanthemum lavandulifoliumChrysanthemum lavandulifoliumt0
Treatment: 5-azacytidine treatmente0
Outcome: resistancee0
Direction: enhancinge0
Comparator: tetraploid phenotypee0

Verbatim

e0
“Chemical inhibition of DNA methylation (5-azacytidine treatment) in diploid plants mimics the tetraploid phenotype by activating WRKY103 expression and enhancing resistance.”
✓ located verbatim in the source · abstract, characters 755–928

t0
“in Chrysanthemum lavandulifolium”
✓ located verbatim in the source · abstract, characters 246–278

Title

Polyploidization enhances plant resistance to Alternaria alternata via DNA hypomethylation activated WRKYs

Zhongyu Yu; Huiting Ci; Ruyue Jing; et al. Horticulture Research 2026. doi:10.1093/hr/uhag050

Other claims from this paper (6)

Intervention effect Autotetraploidization increased Alternaria resistance.
Observation Polyploidization caused promoter CHH hypomethylation in WRKY factors.
Mechanism WRKY transcription was activated upon fungal infection.
Mechanism CIWRKY103 was important for disease resistance.
Intervention effect 5-azacytidine activated WRKY103 expression in diploids.
Observation The epigenetic mechanism was conserved across chrysanthemum species.

Observationobservedoriginal resultmeasured

Claim · model-written summary

Genome analysis identified 49 ATGs in Chrysanthemum lavandulifolium.

Taxon: Chrysanthemum lavandulifoliumChrysanthemum lavandulifoliume0
Outcome: 49e0
Value: 49e0

Verbatim

e0
“49 in Chrysanthemum lavandulifolium”
✓ located verbatim in the source · abstract, characters 663–698

Title

Genome-Wide Identification of Autophagy-Related Gene Family and Gene Expression Analysis of the CmATG8 Under Heat Stress in Chrysanthemum

Bing-Yu Luo; Hui Meng; Zheng-Yu Lu; et al. International Journal of Molecular Sciences 2025. doi:10.3390/ijms26178642

Other claims from this paper (6)

Observation Genome analysis identified 130 ATGs in C. morifolium.
Observation Genome analysis identified 51 ATGs in Chrysanthemum nankingense.
Observation Genes in the same subfamily had conserved structures and domains.
Mechanism Fragment duplication contributed to ATG expansion.
Observation ATG8 genes were the most highly expressed and showed tissue specificity.
Intervention effect Heat stress induced ATG8 transcription, peaking at 48 h.

Observationobservedoriginal resultmeasured

Claim · model-written summary

ClBRN1 belongs to the NAC transcription factor family.

Taxon: Chrysanthemum lavandulifoliumChrysanthemum lavandulifoliumt0
Gene: ClBRN1e0
Outcome: member of the NAC TFs familye0

Not reportedDirection

Verbatim

e0
“the ClBRN1 gene was a member of the NAC TFs family”
✓ located verbatim in the source · abstract, characters 742–792

t0
“Chrysanthemum lavandulifolium”
✓ located verbatim in the source · title, characters 12–41

Title

ClBRN1 from Chrysanthemum lavandulifolium enhances the stress resistance of transgenic Arabidopsis

Yanxi Li; Wenting He; Yueyue Liu; et al. PeerJ 2024. doi:10.7717/peerj.18620

Other claims from this paper (7)

Observation ClBRN1 has a CDS length of 1,080 bp.
Observation ClBRN1 encodes 359 amino acids.
Observation ClBRN1 localized to the nucleus and cell membrane.
Intervention effect ClBRN1 transgenics were significantly shorter.
Intervention effect ClBRN1 improved salt tolerance in Arabidopsis.
Intervention effect ClBRN1 improved low-temperature tolerance in Arabidopsis.
Intervention effect Overexpressing Arabidopsis stems had significantly thicker secondary cell walls than wild type.

Observationobservedoriginal resultmeasured

Claim · model-written summary

ClBRN1 has a CDS length of 1,080 bp.

Taxon: Chrysanthemum lavandulifoliumChrysanthemum lavandulifoliumt0
Gene: ClBRN1t0
Outcome: CDS (coding sequence) lengthe0
Value: 1,080 bpe0

Verbatim

e0
“with a CDS (coding sequence) length of 1,080 bp”
✓ located verbatim in the source · abstract, characters 794–841

t0
“ClBRN1 from Chrysanthemum lavandulifolium”
✓ located verbatim in the source · title, characters 0–41

Title

ClBRN1 from Chrysanthemum lavandulifolium enhances the stress resistance of transgenic Arabidopsis

Yanxi Li; Wenting He; Yueyue Liu; et al. PeerJ 2024. doi:10.7717/peerj.18620

Other claims from this paper (7)

Observation ClBRN1 belongs to the NAC transcription factor family.
Observation ClBRN1 encodes 359 amino acids.
Observation ClBRN1 localized to the nucleus and cell membrane.
Intervention effect ClBRN1 transgenics were significantly shorter.
Intervention effect ClBRN1 improved salt tolerance in Arabidopsis.
Intervention effect ClBRN1 improved low-temperature tolerance in Arabidopsis.
Intervention effect Overexpressing Arabidopsis stems had significantly thicker secondary cell walls than wild type.

Observationobservedoriginal resultmeasured

Claim · model-written summary

ClBRN1 encodes 359 amino acids.

Taxon: Chrysanthemum lavandulifoliumChrysanthemum lavandulifoliumt0
Gene: ClBRN1t0
Outcome: amino acidse0
Value: 359 amino acidse0

Verbatim

e0
“encoding 359 amino acids”
✓ located verbatim in the source · abstract, characters 846–870

t0
“ClBRN1 from Chrysanthemum lavandulifolium”
✓ located verbatim in the source · title, characters 0–41

Title

ClBRN1 from Chrysanthemum lavandulifolium enhances the stress resistance of transgenic Arabidopsis

Yanxi Li; Wenting He; Yueyue Liu; et al. PeerJ 2024. doi:10.7717/peerj.18620

Other claims from this paper (7)

Observation ClBRN1 belongs to the NAC transcription factor family.
Observation ClBRN1 has a CDS length of 1,080 bp.
Observation ClBRN1 localized to the nucleus and cell membrane.
Intervention effect ClBRN1 transgenics were significantly shorter.
Intervention effect ClBRN1 improved salt tolerance in Arabidopsis.
Intervention effect ClBRN1 improved low-temperature tolerance in Arabidopsis.
Intervention effect Overexpressing Arabidopsis stems had significantly thicker secondary cell walls than wild type.