Claim · model-written summary
The nd1 mutant was sensitive to salt stress.
- Taxon
- rice
Oryza sativat0 - Material
- nd1e0
- Treatment
- salt stresse0
- Gene
- OsCSLD4e0
- Outcome
- sensitive to salt stresse0
Not reportedCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingDirectionValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“the OsCSLD4 function-disrupted mutant nd1 was sensitive to salt stress, but insensitive to abscisic acid (ABA)”
t0“rice”
Title
Hui Zhao; Zixuan Li; Yayun Wang; et al. Plant Biotechnology Journal 2021. doi:10.1111/pbi.13729
Other claims from this paper (13)
- Observation The nd1 mutant was insensitive to ABA.
- Observation ABA synthesis and response gene expression was repressed in nd1 under normal and salt conditions.
- Intervention effect Exogenous ABA restored impaired salt tolerance in nd1.
- Intervention effect OsCSLD4 overexpression enhanced rice ABA synthesis gene expression.
- Intervention effect OsCSLD4 overexpression increased rice ABA content.
- Intervention effect OsCSLD4 overexpression improved rice salt tolerance.
- Mechanism The authors inferred that OsCSLD4 regulation of salt tolerance is mediated by ABA synthesis.
- Intervention effect nd1 decreased rice osmotic stress tolerance.
- Intervention effect nd1 did not decrease rice ion toxic tolerance.
- Observation More osmotic-responsive genes than salt-responsive genes were impaired in nd1.
- Mechanism The authors inferred an ABA-induced osmotic response pathway for OsCSLD4-associated rice salt response.
- Intervention effect Disrupting OsCSLD4 reduced grain width and weight.
- Intervention effect OsCSLD4 overexpression increased grain width and weight.