Claim · model-written summary
F. oxysporum infection regulated GIGANTEA (GI) transcription.
- Taxon
- Arabidopsis thaliana
Arabidopsis thalianat0 - Interactor
- F. oxysporum
_(Fusarium_oxysporum_f.sp._batatas)_1234227-PPT-Clemson_University.jpg?width=60)
Fusarium oxysporume0pathogen - Gene
- GIGANTEA (GI)e0
- Outcome
- regulates transcriptione0
Not reportedMaterialCompoundOrgan / stageSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
_(Fusarium_oxysporum_f.sp._batatas)_1234227-PPT-Clemson_University.jpg?width=500)
Verbatim
e0“We report that F. oxysporum infection accelerates flowering time and regulates transcription of a number of floral integrator genes, including FLOWERING LOCUS C (FLC), FLOWERING LOCUS T (FT) and GIGANTEA (GI).”
t0“Investigating the Association between Flowering Time and Defense in the Arabidopsis thaliana-Fusarium oxysporum Interaction”
Title
Rebecca Lyons; Anca Rusu; Jiri Stiller; et al. PLoS ONE 2015. doi:10.1371/journal.pone.0127699
Other claims from this paper (9)
- Intervention effect F. oxysporum infection accelerated flowering.
- Observation F. oxysporum infection regulated FLOWERING LOCUS C (FLC) transcription.
- Observation F. oxysporum infection regulated FLOWERING LOCUS T (FT) transcription.
- Association Late flowering correlated positively with Fusarium resistance in natural ecotypes.
- Observation gi showed enhanced resistance to F. oxysporum.
- Observation autonomous pathway mutants showed enhanced resistance to F. oxysporum.
- Intervention effect FLC deletion accelerated flowering in fve-3 but did not alter disease resistance.
- Intervention effect FLC deletion accelerated flowering in fpa-7 but did not alter disease resistance.
- Mechanism Altered defense gene transcription likely explains fve-3 resistance to F. oxysporum.