Claim · model-written summary
Auxin maxima decrease in Arabidopsis triple sac mutants.
- Taxon
- Arabidopsis
Arabidopsist0 - Material
- triple sac mutantse0
- Outcome
- Auxin maximae0
- Direction
- decreasede0
Not reportedGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“Auxin maxima are decreased in double and triple sac mutants.”
t0“Arabidopsis”
Title
Lizhen Song; Yanning Wang; Zhiai Guo; et al. New Phytologist 2021. doi:10.1111/nph.17402
Other claims from this paper (13)
- Observation The Arabidopsis ncp2 mutant lacked cotyledons in seedlings and embryos in the pid background.
- Observation Arabidopsis NCP2 encodes RHD4/SAC7 phosphoinositide phosphatase.
- Observation SAC6, SAC7 and SAC8 show overlapping and specific expression patterns in Arabidopsis seedlings and embryos.
- Observation Arabidopsis sac6 sac7 embryos either fail to become seeds or have three or four cotyledons.
- Observation Embryo development is significantly delayed or lethal in Arabidopsis sac7 sac8 mutants.
- Observation Embryo development is significantly delayed or lethal in Arabidopsis sac6 sac7 sac8 mutants.
- Observation Arabidopsis sac7 sac8 seedlings are arrested early.
- Observation Arabidopsis sac6 sac7 sac8 seedlings are arrested early.
- Observation Auxin maxima decrease in Arabidopsis double sac mutants.
- Observation PtdIns4P and PtdIns(4,5)P2 contents dramatically increase in Arabidopsis sac6 sac7 mutants.
- Observation PtdIns4P and PtdIns(4,5)P2 contents dramatically increase in Arabidopsis sac7 sac8 mutants.
- Observation PIN1 and PIN2 trafficking from the trans-Golgi network/early endosome back to the plasma membrane is delayed in Arabidopsis sac7 sac8 mutants.
- Mechanism SAC6-SAC8 are essential for phosphoinositide homeostasis and auxin-mediated Arabidopsis development.