Claim · model-written summary
GA-deficient, GA-insensitive, and gamyb mutants had common tapetal PCD defects.
- Taxon
- rice
Oryza sativat0 - Material
- mutantse0
- Organ / stage
- tapetal cellse0
- Outcome
- defects in programmed cell death (PCD) of tapetal cellse0
Not reportedGeneCompoundInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“The mutants exhibited common defects in programmed cell death (PCD) of tapetal cells”
t0“Rice”
Title
Gibberellin Modulates Anther Development in Rice via the Transcriptional Regulation of GAMYB
Koichiro Aya; Miyako Ueguchi-Tanaka; Maki Kondo; et al. The Plant Cell 2009. doi:10.1105/tpc.108.062935
Other claims from this paper (7)
- Observation The same mutants had defects in exine and Ubisch body formation.
- Association GAMYB was involved in nearly all GA-regulated gene expression in rice anthers.
- Observation GAMYB interacted with promoter binding motifs in vitro.
- Intervention effect Mutation of promoter motifs reduced GUS expression in anthers.
- Intervention effect CYP703A3 knockout had gamyb-like defects in exine and Ubisch body formation.
- Mechanism The results suggest GA regulates exine formation and tapetal PCD.
- Mechanism The results suggest direct CYP703A3 activation by GAMYB is key to exine formation.