Claim · model-written summary
Intact probable HrpR was annotated in P. syringae pv. syringae isolates.
- Taxon
- sweet cherry
Prunus aviumt0 - Interactor
- P. syringae pv. syringae
Pseudomonas syringae pv. syringaee0pathogen - Gene
- HrpRe0
- Outcome
- Intact probable regulatory protein HrpRe0
Not reportedMaterialCompoundOrgan / stageSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“Intact probable regulatory protein HrpR was annotated in the genomic sequences of all isolates of P. syringae pv. syringae , P. syringae , P. cerasi , and A .”
t0“sweet cherry”
Title
Tawanda E. Maguvu; Rosa J. Frias; Alejandro I. Hernandez-Rosas; et al. Microbiology Spectrum 2024. doi:10.1128/spectrum.01324-24
Other claims from this paper (18)
- Observation Fifty-eight cherry isolates fell within the P. syringae complex and five genomospecies.
- Observation Intact probable HrpR was annotated in P. syringae isolates.
- Observation Intact probable HrpR was annotated in P. cerasi isolates.
- Observation Intact probable HrpR was annotated in genomospecies A isolates.
- Observation P. viridiflava isolates had atypical probable HrpR.
- Observation Syringomycin and syringopeptin genes were found in the other genomospecies but not A or P. viridiflava.
- Observation P. syringae pv. syringae caused cankers, leaf spots and fruit lesions in the field.
- Observation P. syringae isolates caused only cankers in the field.
- Observation P. cerasi isolates caused only cankers.
- Observation Some P. viridiflava isolates caused only cankers.
- Observation Genomospecies A caused no field symptoms.
- Observation All five genomospecies produced dark lesions on detached immature cherry fruit.
- Association Genomospecies A lesions were significantly less severe than those of other genomospecies.
- Observation P. cerasi was reported as present and pathogenic on California sweet cherry.
- Observation P. viridiflava was reported as present and pathogenic on California sweet cherry.
- Observation Copper resistance in P. syringae pv. syringae was 47.5%.
- Observation No isolates were resistant to kasugamycin.
- Recommendation The authors conclude copper cannot effectively control bacterial blast and canker of sweet cherry.