Claim · model-written summary
Sustained excess H2O2 caused tissue damage in Cat1AS tobacco.
- Taxon
- tobacco
Nicotiana tabacumt0 - Material
- Cat1ASe0
- Treatment
- excess H2O2e0
- Outcome
- tissue damagee0
Not reportedGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingDirectionValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“Sustained exposure of Cat1AS plants to excess H2O2 provoked tissue damage”
t0“tobacco”
Title
Defense activation and enhanced pathogen tolerance induced by H 2 O 2 in transgenic tobacco
Sangpen Chamnongpol; Hilde Willekens; Wolfgang Moeder; et al. Proceedings of the National Academy of Sciences 1998. doi:10.1073/pnas.95.10.5818
Other claims from this paper (10)
- Intervention effect Sustained excess H2O2 stimulated salicylic acid and ethylene production.
- Intervention effect Sustained excess H2O2 induced acidic and basic PR proteins.
- Observation Salicylic acid production was biphasic, with its first peak in the first hours of high light.
- Observation Ethylene peaked in the first hours of high light before tissue necrosis.
- Observation Acidic PR proteins accumulated systemically in upper leaves unexposed to high light.
- Intervention effect Short excess H2O2 exposure did not cause damage.
- Intervention effect Short excess H2O2 exposure locally induced acidic and basic PR proteins.
- Intervention effect Short excess H2O2 exposure enhanced pathogen tolerance.
- Observation PR protein induction after short exposure resembled upper uninfected leaves more than hypersensitive-response leaves.
- Association Rapid and strong PR expression activation occurred only when excess H2O2 accompanied leaf necrosis.