Claim · model-written summary
The authors suggest that fungal ABA could trigger host ABA signaling at infection sites.
- Taxon
- Rice
Oryza sativat0 - Interactor
- blast-funguse0pathogen
- Compound
- ABAe0
- Outcome
- ABA signaling at host infection sitese0
- Qualifier
- could play a rolee0
Not reportedMaterialGeneOrgan / stageSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValue
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“blast-fungus-derived ABA could play a role in triggering ABA signaling at host infection sites.”
t0“Rice–Magnaporthe griseaInteraction”
Title
Chang-Jie Jiang; Masaki Shimono; Shoji Sugano; et al. Molecular Plant-Microbe Interactions 2010. doi:10.1094/mpmi-23-6-0791
Other claims from this paper (9)
- Intervention effect Exogenous ABA compromised rice resistance to compatible M. grisea strains.
- Intervention effect ABA suppressed transcriptional upregulation of WRKY45 induced by SA, benzothiadiazole or blast infection.
- Intervention effect ABA suppressed transcriptional upregulation of OsNPR1 induced by SA, benzothiadiazole or blast infection.
- Intervention effect OsNPR1 overexpression largely negated ABA-enhanced blast susceptibility.
- Intervention effect WRKY45 overexpression largely negated ABA-enhanced blast susceptibility.
- Observation ABA-responsive genes were induced reciprocally to WRKY45 and OsPR1b in compatible blast interaction, but only marginally in incompatible interaction.
- Observation ABA was detected in M. grisea hyphae and conidia and in culture media.
- Mechanism The authors infer ABA acts upstream of WRKY45 and OsNPR1 in rice SA signaling.
- Mechanism The authors suggest that the SA–ABA signaling balance determines rice–M. grisea interaction outcomes.