Claim · model-written summary
Actinomycin D applied with IWF-R prevented resistance induction.
- Taxon
- Phaseolus vulgaris
Phaseolus vulgarise1 - Treatment
- actinomycin De0
- Outcome
- resistance inducing effect of IWF‐Re0
- Direction
- preventede0
Not reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“When the transcription inhibitor actinomycin D and the translation inhibitor cycloheximide were applied simultaneously with IWF‐R 3 dai the resistance inducing effect of IWF‐R was prevented”
e1“bean plants (Phaseolus vulgaris, cv. Pinto)”
Title
B. Kutzner; K. ‐H. Hellwald; H. Buchenauer Journal of Phytopathology 1993. doi:10.1111/j.1439-0434.1993.tb01356.x
Other claims from this paper (16)
- Intervention effect Preinoculation with Uromyces phaseoli induced systemic TNV resistance in bean.
- Observation Systemic resistance required at least four days between primary and challenge inoculation and peaked after six days.
- Intervention effect Rust-infected leaf washing fluid reduced TNV disease incidence in bean.
- Observation Extracts taken six days after rust inoculation induced the strongest resistance effect.
- Observation At least three days between IWF-R application and TNV inoculation was necessary for induced resistance.
- Intervention effect Washing fluid from healthy leaves did not induce TNV resistance.
- Intervention effect IWF-R systemic protection reduced TNV disease by an average of 50%.
- Intervention effect IWF-R retained resistance-inducing capacity after dilution to one-quarter concentration.
- Observation IWF-R and IWF-H inactivated virus locally in treated leaf areas.
- Intervention effect IWF application near TNV inoculation markedly reduced local lesions.
- Association Longer pre- or postinfection intervals significantly diminished IWF activity.
- Association IWF-R efficacy increased at prolonged preinfection intervals, whereas healthy-plant extracts were ineffective.
- Mechanism The biphasic activity pattern suggests IWF-R contains agents that inactivate virus and induce resistance.
- Intervention effect Cycloheximide applied with IWF-R prevented resistance induction.
- Observation Preliminary ultrafiltration limited the size of IWF-R resistance-inducing ingredients to 10–20 kd.
- Observation Preliminary ultrafiltration limited the size of virus-inactivating compounds to 1–5 kd.