Claim · model-written summary
LbMUG3 delayed salt gland development.
- Taxon
- L. bicolorLimonium bicolore0
- Gene
- LbMUG3e0
- Outcome
- salt gland developmente0
- Direction
- delayede0
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingValueQualifier
Verbatim
e0“LbMUG3 delayed salt gland development in L. bicolor”
Title
Jianglu Zhu; Yiyang Feng; Hai Fan; et al. Annals of Botany 2026. doi:10.1093/aob/mcag036
Other claims from this paper (10)
- Observation LbSTI was specifically expressed in Limonium bicolor salt glands.
- Intervention effect LbSTI overexpression increased salt gland number in Limonium bicolor.
- Intervention effect LbSTI overexpression increased salt secretion in Limonium bicolor.
- Intervention effect LbSTI overexpression increased salinity tolerance in Limonium bicolor.
- Intervention effect LbSTI silencing produced opposite salt gland, secretion, and tolerance phenotypes.
- Intervention effect LbSTI increased trichome development in Arabidopsis wild type and sti mutant plants.
- Intervention effect LbSTI increased salinity tolerance in Arabidopsis wild type and sti mutant plants.
- Observation LbMUG3 bound the LbSTI promoter.
- Mechanism LbMUG3 inhibited LbSTI transcription.
- Intervention effect Silencing LbMUG3 upregulated LbSTI expression.