Claim · model-written summary
The C129S mutation prevented rescue of leaf serration defects in ibr5-3.
- Material
- ibr5-3 mutante0
- Treatment
- Introducing a C129S mutatione0
- Gene
- IBR5e0
- Outcome
- leaf-serration defectse0
- Direction
- unable to rescuee0
Not reportedTaxonCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Verbatim
e0“Introducing a C129S mutation within the highly conserved VxVHCx2GxSRSx5AYLM motif of IBR5 rendered it unable to rescue the leaf-serration defects of the ibr5-3 mutant.”
Title
IBR5 Regulates Leaf Serrations Development via Modulation of the Expression of PIN1
Xiuzhen Kong; Guoqiang Huang; Yali Xiong; et al. International Journal of Molecular Sciences 2019. doi:10.3390/ijms20184429
Other claims from this paper (5)
- Observation IBR5 loss-of-function mutants showed pronounced serrations associated with increased cell area.
- Observation IBR5 localized in nuclei of leaf epidermis and petiole cells.
- Observation Auxin maxima distribution expanded ectopically in ibr5-3.
- Observation PIN1 distribution on the plasma membrane was compromised in ibr5-3.
- Mechanism The authors conclude IBR5 is essential to establish PIN1-directed auxin maxima at leaf serration tips.