Claim · model-written summary
Membrane depolarization terminated repetitive cytosolic calcium elevations.
- Taxon
- Vicia faba
Vicia fabat0 - Treatment
- Depolarization of the membranee0
- Outcome
- repetitive elevations in cytosolic Ca2+e0
- Direction
- terminatede0
Not reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“Depolarization of the membrane terminated both repetitive elevations in cytosolic Ca2+ and inward-directed ion currents”
t0“Vicia faba guard cells”
Title
J I Schroeder; S Hagiwara Proceedings of the National Academy of Sciences 1990. doi:10.1073/pnas.87.23.9305
Other claims from this paper (9)
- Observation Resting guard-cell cytosolic calcium was 0.19 +/- 0.09 microM.
- Intervention effect ABA exposure caused transient repetitive increases in cytosolic free calcium in responsive guard cells.
- Association Calcium transients coincided with increases in inward ion current.
- Intervention effect Membrane depolarization terminated inward ion currents.
- Mechanism The authors infer ABA calcium transients arise from passive extracellular calcium influx through permeable channels.
- Observation ABA activated currents reversed at depolarizations above -10 mV.
- Observation ABA-induced currents were not highly calcium selective and permitted calcium and potassium permeation.
- Mechanism ABA activated calcium permeable channels in the guard-cell plasma membrane.
- Mechanism The authors infer calcium elevations can modulate responses that promote stomatal closure.