Claim · model-written summary
The authors conclude that the TCV coat protein is the viral determinant of resistance of Arabidopsis Di-0 to TCV.
- Taxon
- Arabidopsis
Arabidopsist0 - Material
- Di-0e2
- Interactor
- TCVe2pathogen
- Gene
- TCV coat proteine2
- Outcome
- resistance of Di-0 to TCVe2
Not reportedCompoundOrgan / stageSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
t0“Arabidopsis”
e2“These results indicate that TCV coat protein is the viral determinant for resistance of Di-0 to TCV”
Title
Qingzhong Kong ScholarWorks@UMassAmherst (University of Massachusetts Amherst) 1996
Other claims from this paper (12)
- Observation TCV variants carrying the CCFV coat protein ORF or a coat protein start-codon mutation (TCV-CPm) were infectious on the susceptible ecotype Col-0 of Arabidopsis.
- Observation TCV variants carrying the CCFV coat protein ORF or a coat protein start-codon mutation (TCV-CPm) were infectious on the TCV-resistant ecotype Di-0 of Arabidopsis.
- Observation The authors suggest that the resistance of Di-0 to TCV is specific to TCV.
- Mechanism The authors suggest that the resistance of Di-0 to TCV may involve an RNA degradation activity.
- Observation TCV was restricted to small areas around the initial infection sites on inoculated leaves of Di-0 plants, as shown by whole plant in situ hybridization.
- Observation TCV in Col-0 plants moved long-distance to metabolic sink tissues (young leaves and roots) at a rate that differed from the other viruses tested.
- Observation CCFV moved long-distance to metabolic sink tissues (young leaves and roots) at a rate that differed from the other viruses tested.
- Mechanism Symptom modulation by sat-RNA C in Arabidopsis is mediated by the viral coat protein.
- Mechanism Inhibition of virus long distance movement is involved in sat-RNA C-mediated symptom attenuation of TCV-CPm.
- Mechanism Inhibition of helper virus replication is not important for sat-RNA C-mediated symptom attenuation.
- Observation Symptom attenuation mediated by sat-RNA C is localized.
- Mechanism Symptom attenuation mediated by sat-RNA C does not involve the major plant defense pathway.