Claim · model-written summary
Mutant and wild-type viruses had similar rootlet virus content after four weeks.
- Taxon
- sugar beet
Beta vulgaris subsp. vulgarist0 - Treatment
- mutante0
- Interactor
- beet necrotic yellow vein virus
Beet necrotic yellow vein virust0pathogen - Outcome
- virus content in rootletse0
- Direction
- No differencese0
- Comparator
- wild-type virusese0
Not reportedMaterialGeneCompoundOrgan / stageSecond interactorSecond interactor roleSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“No differences were found in virus content in rootlets between mutant and wild-type viruses or between susceptible and resistant cultivars after culture for 4 weeks in a growth cabinet”
t0“Beet Necrotic Yellow Vein Virus Do Not Cause Rhizomania Disease in Sugar Beets”
Title
Tetsuo Tamada; Hirokatsu Uchino; Toshimi Kusume; et al. Phytopathology 1999. doi:10.1094/phyto.1999.89.11.1000
Other claims from this paper (9)
- Observation Susceptible and resistant cultivars had similar rootlet virus content after four weeks.
- Intervention effect Wild-type virus caused rhizomania symptoms and 69 to 96% yield loss in susceptible cultivars in the field.
- Observation Most resistant-cultivar plants had no symptoms but had 23% yield loss.
- Observation Resistant plants had 10 to 20x lower root virus concentrations than susceptible plants.
- Intervention effect Mutant strains caused no symptoms in susceptible or resistant cultivars.
- Intervention effect Mutant infection produced much lower root virus content than wild-type infection in both cultivars.
- Observation Wild-type RNA 3 was undetectable in most symptomless resistant taproots.
- Mechanism P25 of viral RNA 3 is essential for rhizomania symptoms in susceptible cultivars.
- Mechanism P25 may fail to facilitate virus movement from rootlets to taproots in partially resistant plants.