Claim · model-written summary
P25 may fail to facilitate virus movement from rootlets to taproots in partially resistant plants.
- Taxon
- sugar beet
Beta vulgaris subsp. vulgarist0 - Gene
- P25e0
- Outcome
- virus translocation from rootlets to taprootse0
- Qualifier
- may fail to facilitatee0
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValue
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“These results indicate that the P25 of BNYVV RNA 3 is essential for the development of rhizomania symptoms in susceptible cultivars and suggest that it may fail to facilitate virus translocation from rootlets to taproots in the partially resistant cultivar”
t0“Beet Necrotic Yellow Vein Virus Do Not Cause Rhizomania Disease in Sugar Beets”
Title
Tetsuo Tamada; Hirokatsu Uchino; Toshimi Kusume; et al. Phytopathology 1999. doi:10.1094/phyto.1999.89.11.1000
Other claims from this paper (9)
- Intervention effect Mutant and wild-type viruses had similar rootlet virus content after four weeks.
- Observation Susceptible and resistant cultivars had similar rootlet virus content after four weeks.
- Intervention effect Wild-type virus caused rhizomania symptoms and 69 to 96% yield loss in susceptible cultivars in the field.
- Observation Most resistant-cultivar plants had no symptoms but had 23% yield loss.
- Observation Resistant plants had 10 to 20x lower root virus concentrations than susceptible plants.
- Intervention effect Mutant strains caused no symptoms in susceptible or resistant cultivars.
- Intervention effect Mutant infection produced much lower root virus content than wild-type infection in both cultivars.
- Observation Wild-type RNA 3 was undetectable in most symptomless resistant taproots.
- Mechanism P25 of viral RNA 3 is essential for rhizomania symptoms in susceptible cultivars.