Claim · model-written summary
SA-induced Hsp17.6 expression had a different dose response from PR-1 expression.
- Taxon
- Arabidopsis thaliana
Arabidopsis thalianat0 - Treatment
- SAe0
- Outcome
- Hsp17.6 expressione0
- Direction
- differente0
- Comparator
- PR-1 expressione0
Not reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“SA-induced Hsp17.6 expression had a different dose-response to PR-1 expression.”
t0“Arabidopsis thaliana”
Title
Shannon M. Clarke; Luis A. J. Mur; Jacqueline E. Wood; et al. The Plant Journal 2004. doi:10.1111/j.1365-313x.2004.02054.x
Other claims from this paper (12)
- Association SA-dependent signaling contributed to basal thermotolerance from seeds through three-week-old plants.
- Association Endogenous SA correlated with basal thermotolerance; NahG had the lowest and cpr5 the highest tolerance.
- Intervention effect SA promoted thermotolerance during heat shock and recovery.
- Mechanism Recovery from heat shock apparently involved an NPR1-dependent pathway.
- Mechanism Thermotolerance during heat shock did not involve an NPR1-dependent pathway.
- Intervention effect SA reduced electrolyte leakage.
- Intervention effect SA or heat shock induced PR-1 and Hsp17.6.
- Observation Heat-shock-induced Hsp17.6 protein appeared more slowly in NahG.
- Intervention effect SA only partially induced HSPs.
- Intervention effect Heat shock induced Hsp17.6 more substantially than SA.
- Intervention effect SA had no effect on Hsp101 expression.
- Intervention effect All genotypes expressed HSPs and acquired heat-shock tolerance after prior heat acclimation.