Claim · model-written summary
Sclerotinia infection induced BnaWRKY75 more strongly than other WRKY genes.
- Interactor
- S. sclerotiorum
Sclerotinia sclerotiorume0pathogen - Gene
- BnaWRKY75e0
- Outcome
- WRKY genee0
- Direction
- most highly inducede0
Not reportedTaxonMaterialCompoundOrgan / stageSecond interactorSecond interactor roleTreatmentComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“BnaWRKY75 was the most highly induced WRKY gene upon S. sclerotiorum infection”
Title
Dongxiao Liu; Jialin Fan; Yi Ye; et al. Journal of Experimental Botany 2025. doi:10.1093/jxb/eraf332
Other claims from this paper (10)
- Mechanism BnaWRKY75 positively regulates resistance to Sclerotinia sclerotiorum.
- Intervention effect BnaA10.WRKY75 overexpression enhanced Arabidopsis resistance to Sclerotinia.
- Intervention effect BnaA10.WRKY75 overexpression enhanced oilseed rape resistance to Sclerotinia.
- Intervention effect BnaWRKY75 knockout reduced resistance.
- Association Sequencing implicated BnaICS1 as a possible BnaA10.WRKY75 target.
- Mechanism BnaA10.WRKY75 binds the BnaCnn.ICS1 promoter and activates its transcription.
- Mechanism BnaA10.WRKY75 promoted SA biosynthesis and SA-dependent defense.
- Mechanism Arabidopsis genetic analysis placed ICS1 downstream of BnaA10.WRKY75.
- Intervention effect Salicylic acid treatment significantly enhanced oilseed rape resistance to Sclerotinia.
- Observation Oilseed rape BnaICS1 homologues differed in transcriptional regulation.