Claim · model-written summary
ParA1 treatment induced SlSOBIR1 relocalization to endosomal compartments.
- Taxon
- Tomato
Solanum lycopersicumt0 - Treatment
- elicitin (ParA1) treatmente0
- Gene
- SlSOBIR1e0
- Outcome
- relocalization of SlSOBIR1 from the plasma membrane to endosomal compartmentse0
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingDirectionValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“P. parasitica infection and elicitin (ParA1) treatment induced the relocalization of SlSOBIR1 from the plasma membrane to endosomal compartments”
t0“Tomato”
Title
Ke-Chun Peng; Chao-Wen Wang; Chih-Hang Wu; et al. Molecular Plant-Microbe Interactions 2015. doi:10.1094/mpmi-12-14-0405-r
Other claims from this paper (5)
- Intervention effect Silencing SlSOBIR1 and SlSOBIR1-like enhanced tomato susceptibility to P. parasitica.
- Intervention effect Silenced plants had compromised callose deposition, ROS production and PTI marker expression.
- Observation P. parasitica infection induced SlSOBIR1 relocalization to endosomal compartments.
- Intervention effect NbSOBIR1 silencing compromised ParA1-mediated cell death in Nicotiana benthamiana.
- Mechanism The SlSOBIR1 kinase domain was indispensable for ParA1-triggered internalization and cell death.