Claim · model-written summary
CsMYB44 represses expression of its four target genes.
- Taxon
- Camellia sinensis
Camellia sinensist0 - Gene
- CsMYB44e0
- Outcome
- their expressione0
- Direction
- represse0
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“CsMYB44 directly binds to the promoters of CsICE1, CsCHS (chalcone synthase), CsFLS (flavonol synthase), and CsANR (anthocyanidin reductase) to repress their expression.”
t0“Camellia sinensis”
Title
Chenyu Shao; Qian Zhu; Rong Xu; et al. The Plant Journal 2026. doi:10.1111/tpj.70737
Other claims from this paper (14)
- Association CsICE1 positively correlated with catechin accumulation and cold tolerance.
- Association CsMYB44 negatively correlated with catechin accumulation and cold tolerance.
- Intervention effect CsICE1 overexpression improved Arabidopsis cold tolerance.
- Intervention effect CsICE1 overexpression elevated Arabidopsis flavonoid levels.
- Intervention effect CsICE1 overexpression upregulated Arabidopsis antioxidant and cold responsive genes.
- Intervention effect Changing CsICE1 expression significantly affected cold sensitivity in tea.
- Intervention effect CsMYB44 overexpression reduced Arabidopsis cold tolerance.
- Intervention effect Silencing CsMYB44 enhanced tea cold tolerance.
- Observation CsMYB44 directly binds promoters of CsICE1, CsCHS, CsFLS and CsANR.
- Mechanism CsICE1 activates transcription of CsCHS, CsFLS and CsANR.
- Intervention effect Short term cold stress suppresses CsMYB44 expression.
- Mechanism Cold induced CsMYB44 suppression releases CsICE1.
- Mechanism Released CsICE1 promotes flavonoid and catechin accumulation through target gene upregulation.
- Mechanism The CsICE1 pathway mitigates cold induced damage.