Claim · model-written summary
NbCRT3a silencing significantly compromised resistance to P. infestans.
- Taxon
- Nicotiana benthamiana
Nicotiana benthamianat0 - Treatment
- NbCRT3a-silencede1
- Interactor
- P. infestans
Phytophthora infestanse0pathogen - Outcome
- resistance to P. infestanse0
- Direction
- compromisede0
Not reportedMaterialGeneCompoundOrgan / stageSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“resistance to P. infestans was significantly compromised.”
e1“NbCRT3a-silenced plants showed no detectable growth defects but resistance”
t0“Nicotiana benthamiana”
Title
Mizuki Matsukawa; Yusuke Shibata; Mina Ohtsu; et al. Molecular Plant-Microbe Interactions 2013. doi:10.1094/mpmi-12-12-0301-r
Other claims from this paper (12)
- Observation Mature Nicotiana benthamiana strongly resisted P. infestans.
- Method / resource The silencing screen identified NbCRT3a as required for resistance against P. infestans.
- Mechanism NbCRT3a encodes an ER quality-control chaperone for glycoprotein maturation.
- Intervention effect NbCRT3a silencing caused no detectable growth defects.
- Intervention effect NbCRT3a silencing suppressed INF1-induced reactive oxygen species production.
- Intervention effect NbCRT3a silencing suppressed INF1-induced phytoalexin accumulation.
- Intervention effect NbCRT3a silencing reduced NbEAS expression.
- Intervention effect NbCRT3a silencing did not affect NbPR-1a expression.
- Intervention effect NbCRT3a silencing suppressed INF1-induced ethylene production.
- Intervention effect NbCRT3a silencing impaired resistance reactions induced by P. infestans wall components.
- Intervention effect NbCRT3a silencing did not affect cell death induced by active NbMEK2(DD).
- Mechanism NbCRT3a was inferred to be necessary to initiate resistance to P. infestans elicitors.