Claim · model-written summary
Smooth-stem wound resistance persisted despite salicylic acid, ethylene, jasmonate or abscisic acid signaling disruption.
- Taxon
- tomato
Solanum lycopersicumt0 - Outcome
- resistance of smooth stem woundse0
- Direction
- could not be alterede0
- Qualifier
- disruption of salicylic acid, ethylene, jasmonate or abscisic acid signallinge0
Not reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingValue
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“resistance of smooth stem wounds could not be altered by disruption of salicylic acid, ethylene, jasmonate or abscisic acid signalling”
t0“tomato”
Title
T. Beyers; C. Vos; R. Aerts; et al. Plant Pathology 2013. doi:10.1111/ppa.12075
Other claims from this paper (9)
- Intervention effect Flush pruning gave tomato stem wounds absolute resistance.
- Observation Petiole stubs were highly susceptible to B. cinerea.
- Observation Wound resistance became nearly complete within 48 hours after deleafing.
- Mechanism Wound drying did not explain the decline in susceptibility.
- Association Disease-signaling mutants showed altered susceptibility to B. cinerea.
- Mechanism Ethylene signaling was implicated in petiole-stub defense.
- Association Jasmonate-biosynthesis mutants had decreased susceptibility.
- Association Abscisic-acid-biosynthesis mutants had decreased susceptibility.
- Mechanism The authors infer that smooth-wound resistance does not depend on major disease signaling pathways.