Claim · model-written summary
MPF2 controlled sepal cell division in Physalis floridana.
- Taxon
- P. floridana
Physalis pubescense1 - Gene
- MPF2e0
- Outcome
- sepal cell divisione0
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“MPF2 controls sepal cell division”
e1“However, heterotopic expression of MPF2 is not sufficient for ICS formation in P. floridana”
Title
Hormonal control of the inflated calyx syndrome, a morphological novelty, in Physalis
Chaoying He; Heinz Saedler The Plant Journal 2007. doi:10.1111/j.1365-313x.2006.03008.x
Other claims from this paper (11)
- Association MPF2 heterotopic expression is associated with inflated calyx syndrome in Physalis floridana.
- Observation The MPF2 promoter differs significantly from the STMADS16 promoter in Solanum tuberosum.
- Observation MPF2 expression alone was insufficient for inflated calyx formation in Physalis floridana.
- Mechanism Fertilization was important for inflated calyx formation in Physalis floridana.
- Mechanism Cytokinin and gibberellin were essential for inflated calyx formation in Physalis floridana.
- Observation Cells resulting from MPF2 controlled division were small.
- Mechanism Gibberellin and cytokinin promoted elongation, cell division and substantial calyx enlargement.
- Intervention effect Cytokinin facilitated nuclear transport of MPF2 or STMADS16 fusion proteins.
- Intervention effect Ectopic STMADS16 expression with cytokinin and gibberellin induced an ICS like structure in transgenic Solanum tuberosum.
- Intervention effect In transgenic Arabidopsis, solanaceous MPF2 like proteins enhanced sepal growth with cytokinin.
- Observation Non solanaceous orthologous proteins did not require cytokinin for sepal growth in transgenic Arabidopsis.