Claim · model-written summary
ECH loss prolonged endoplasmic reticulum stress chronically.
- Taxon
- Arabidopsis thaliana
Arabidopsis thalianae1 - Treatment
- ECH losse0
- Gene
- ECHe0
- Outcome
- endoplasmic reticulum stresse0
- Direction
- prolongede0
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“ECH loss led to a chronically prolonged endoplasmic reticulum stress in the ech mutant”
e1“in Arabidopsis thaliana”
Title
Lijiang Liu; Li Qin; Luqman Bin Safdar; et al. PLANT PHYSIOLOGY 2022. doi:10.1093/plphys/kiac400
Other claims from this paper (6)
- Intervention effect ECH loss compromised penetration resistance to powdery mildew.
- Intervention effect ECH loss enhanced postinvasion resistance to powdery mildew.
- Mechanism ECH loss caused salicylic acid hyperaccumulation via ISOCHORISMATE SYNTHASE 1 biosynthesis.
- Mechanism SA hyperaccumulation constitutively activated SA-dependent innate immunity, largely accounting for enhanced postinvasion resistance.
- Observation The ech mutant had accelerated spontaneous cell death independent of SA.
- Observation The ech mutant had constitutive callose deposition mediated by POWDERY MILDEW RESISTANCE 4.