Claim · model-written summary
XT37 and XT122 had insertion sites in indigenous plasmids.
- Material
- XT37 and XT122e0
- Interactor
- Xanthomonas axonopodis pv. citri
Xanthomonas citrit0pathogen - Outcome
- XT37 and XT122 were located in the indigenous plasmidse0
CitrusNot reportedGeneCompoundOrgan / stageSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“those for XT37 and XT122 were located in the indigenous plasmids”
t0“citrus canker pathogen Xanthomonas axonopodis pv. citri”
Title
Shu Yun Tung; Tsong Teh Kuo Canadian Journal of Botany 2000. doi:10.1139/b00-078
Other claims from this paper (11)
- Observation XT10 and XT122 caused no visible host symptoms.
- Observation XT27 induced a callus-like lesion but no watersoaked lesion.
- Observation XT37 induced a watersoaked lesion but no callus-like eruption.
- Observation The nonpathogenic mutants failed to grow in citrus tissue.
- Observation XT27 and XT37 grew more slowly in citrus than wild type.
- Intervention effect Co-inoculation of leaves with XT27 and XT37 did not complement their defects.
- Observation Mutants and wild type had similar extracellular polysaccharide, protease and amylase activities.
- Observation XT27 had reduced extracellular polygalacturonate lyase activity.
- Observation XT27 required glutamic acid for growth in culture.
- Observation Each mutant had one transposon insertion site.
- Observation XT10 and XT27 had chromosomal insertion sites.