Claim · model-written summary
Coexpressing AvrPtoB did not inhibit DspA/E-induced cell death in Nicotiana benthamiana.
- Taxon
- Nicotiana benthamiana
Nicotiana benthamianat1 - Treatment
- coexpression of AvrPtoBe2
- Interactor
- Erwinia amylovora
Erwinia amylovorat0pathogen - Second interactor
- Pseudomonas syringae pv. tomato
Pseudomonas syringae pv. tomatoe2pathogen - Outcome
- DspA/E-induced cell deathe2
- Direction
- not inhibitede2
Not reportedMaterialGeneCompoundOrgan / stageComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
t0“a type III effector of Erwinia amylovora”
t1“is required for early rapid growth in Nicotiana benthamiana”
e2“DspA/E-induced cell death in N. benthamiana was not inhibited by coexpression of AvrPtoB of Pseudomonas syringae pv. tomato”
Title
CHANG‐SIK OH; GREGORY B. MARTIN; STEVEN V. BEER Molecular Plant Pathology 2007. doi:10.1111/j.1364-3703.2007.00390.x
Other claims from this paper (8)
- Intervention effect Silencing NbSGT1 prevented DspA/E-induced cell death in Nicotiana benthamiana.
- Intervention effect Silencing NbRAR1 or two MAP kinase kinase genes did not prevent DspA/E-induced cell death in Nicotiana benthamiana.
- Intervention effect Silencing NbSGT1 compromised non-host resistance of Nicotiana benthamiana against Erwinia amylovora.
- Observation Erwinia amylovora grew rapidly within the first 24 h in Nicotiana benthamiana, and DspA/E was required for this growth.
- Observation Bacterial cell numbers decreased after 24 h in TRV-vector-transformed Nicotiana benthamiana plants.
- Observation A dspA/E mutant strain grew to high populations in NbSGT1-silenced Nicotiana benthamiana plants.
- Mechanism Nicotiana benthamiana recognizes the bacteria, resulting in programmed cell death and restriction of bacterial growth.
- Observation DspA/E enhances the virulence of Erwinia amylovora in Nicotiana benthamiana.