Claim · model-written summary
Early infected Japanese birch callus is interpreted as inducing defense proteins related to photosynthesis, energy production and mitochondrial protein import.
Inonotus obliquusVerbatim
e0“The obtained results suggest that Japanese birch callus is capable of inducing several proteins related to photosynthesis, energy production, protein import machinery in mitochondria, protein folding, detoxification, secondary cell wall formation, and PR‐10 protein at the early infection stage as responses to defend itself against I. obliquus strain IO‐U1 infection.”
Title
Retno Agnestisia; Rei Chino; Ikumi Nezu; et al. Forest Pathology 2025. doi:10.1111/efp.70051
Other claims from this paper (6)
- Observation Proteome analysis detected 134 infection-specific protein spots in Japanese birch callus.
- Observation Highly expressed infection-specific proteins included protochlorophyllide reductase chloroplastic, enolase and mitochondrial-processing peptidase subunit alpha-like.
- Observation Identified infection-specific proteins included mitochondrial-processing peptidase subunit alpha isoform B, T-complex protein 1 subunit eta and glutathione S-transferase-like protein.
- Observation Identified infection-specific proteins included partial glutathione S-transferase-like protein, WALLS ARE THIN 1-like and 1 Sc-3.
- Mechanism Early infected birch callus is interpreted as inducing defense proteins related to protein folding, detoxification and secondary cell wall formation.
- Mechanism Early infected birch callus is interpreted as inducing PR-10 protein as a defense response.