Claim · model-written summary
The anti-ripening action of melatonin is reversed by cold, according to the authors.
- Taxon
- sweet cherry
Prunus aviume0 - Outcome
- anti-ripening action of melatonine1
- Qualifier
- reversed by colde1
Not reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValue
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“on ripening parameters of sweet cherry (cv. Ferrovia) fruit”
e1“this anti-ripening action of melatonin is reversed by cold”
Title
Michail Michailidis; Georgia Tanou; Eirini Sarrou; et al. Frontiers in Nutrition 2021. doi:10.3389/fnut.2021.695061
Other claims from this paper (10)
- Intervention effect Preharvest melatonin spray depressed fruit respiration at harvest.
- Intervention effect Combined pre- and postharvest melatonin treatments delayed fruit softening after cold exposure.
- Intervention effect All applied melatonin treatments induced respiratory activity after cold storage.
- Intervention effect Melatonin upregulated the tricarboxylic acid cycle genes PaFUM, PaOGDH and PaIDH in fruit, particularly after combined application.
- Intervention effect Melatonin upregulated the tricarboxylic acid cycle gene PaPDHA1 in fruit, particularly after combined application.
- Intervention effect Melatonin-treated cherries had higher phenolic compound accumulation at harvest and after cold exposure, with the highest effect after combined pre- and postharvest treatment.
- Intervention effect Melatonin raised the expression of PaSK, PaPAL and Pa4CL genes involved in phenol biosynthesis.
- Intervention effect Melatonin raised the expression of the PaC4H gene involved in phenol biosynthesis.
- Intervention effect Melatonin raised the expression of the PaFNR gene involved in phenol biosynthesis.
- Intervention effect Several ripening traits were not influenced by melatonin.