Claim · model-written summary
XopQ enhances bacterial growth in resistant pepper.
Not reportedMaterialGeneCompoundOrgan / stageSecond interactorSecond interactor roleTreatmentComparatorSettingValueQualifier
Verbatim
e0“enhances bacterial growth in resistant pepper and tomato (Solanum lycopersicum)”
t0“Xanthomonas euvesicatoria type III effector XopQ interacts with tomato and pepper 14–3–3 isoforms to suppress effector‐triggered immunity”
e1“this effector inhibits ETI-associated cell death induced by avirulent Xcv in resistant pepper (Capsicum annuum)”
Title
Doron Teper; Dor Salomon; Sukumaran Sunitha; et al. The Plant Journal 2013. doi:10.1111/tpj.12391
Other claims from this paper (13)
- Mechanism XopQ suppresses ETI-associated cell death induced by MAP kinase cascade components and R/avr pairs.
- Mechanism XopQ inhibits ETI-associated cell death in resistant pepper challenged with avirulent Xcv.
- Observation XopQ enhances bacterial growth in resistant tomato.
- Observation Tomato SlTFT4 and homologs interact with XopQ.
- Intervention effect Mutation of the XopQ binding site impaired interaction with CaTFT4 in yeast.
- Intervention effect Mutation of the XopQ binding site impaired virulence in planta.
- Observation TFT4 mRNA abundance increased during incompatible tomato interaction with Xcv.
- Observation TFT4 mRNA abundance increased during incompatible pepper interaction with Xcv.
- Intervention effect Silencing NbTFT4 reduced MAPKKKα-induced cell death in Nicotiana benthamiana.
- Intervention effect CaTFT4 silencing delayed ETI-associated cell death in pepper.
- Intervention effect CaTFT4 silencing enhanced growth of virulent Xcv in pepper.
- Intervention effect CaTFT4 silencing enhanced growth of avirulent Xcv in pepper.
- Mechanism The authors suggest XopQ suppresses ETI and immunity-associated cell death through TFT4 interaction.